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PCs stimulate myocardial mitochondrial phosphorylation, modulating mitochondrial uncoupling rates and affecting respiratory function ( In conclusion, PCs can alleviate diabetes-induced myocardial hypertrophy, cardiac fibrosis, and heart failure, demonstrating potential cardioprotective effects by regulating the S100A11/RAGE/PPAR signaling pathway, improving mitochondrial function, reducing oxidative stress, and inhibiting the formation of AGEs

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Integration of TEs either through cut-and-paste or retrotransposition can happen either inside genes (affecting the sequence of the gene) or in the vicinity of genes (which can lead to abnormal activation or repression of the neighboring genes) [59]

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