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FIGURE 6 To further gain insight into the possible mechanisms underlying the effect of CS on mitochondrial homeostasis, we examined its effect on mitochondrial fission and fusion, which are important processes for functional mitochondria after cell damage (Supplementary Figure S6)

Under iron stimulation, HFE-deficient Kupffer cells phagocytose LDL-C in large amounts and reduce blood lipid levels ( The discovery of ferroptosis may explain the biological effects of iron accumulation

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These findings suggest that a novel, feasible, and effective modality was developed for gastric ulcer gene therapy