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This review synthesizes existing evidence to propose that ferroptosis acts as an important regulatory node within the pathological network of periodontitis, impairs epithelial barrier function, suppresses osteoblast differentiation and function, and amplifies pro-inflammatory signaling pathways through cell-type-specific mechanisms

Psat1-generated -ketoglutarate and glutamine promote muscle stem cell activation and regeneration

Diabetes-induced dysfunction of mitochondria and stem cells in skeletal muscle and the nervous system

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