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Skipping of exon 14 removes Y1003, a key phosphorylation site required for CBL-mediated c-MET ubiquitination and subsequent degradation, resulting in aberrant c-MET accumulation and prolonged signaling, thereby driving unchecked cellular proliferation [45,46,47,48]

For pull-downs, we used two antibodies: against the full phospho-motif sequence of PKD LxRxx[S*/T*] and antibody against part of the PKD phospho-motif sequence Rxx[S*/T*]

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