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Additionally, Magnon et al
TREM2, which plays critical roles in phagocytosis and lipid metabolism, represents another compelling target, particularly as certain TREM2 variants significantly increase AD risk [29, 486, 487]
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However, the proportion of all spines containing non-synaptic GluA1 clusters was significantly lower than that observed for non-synaptic GluA2 clusters (Supplementary Fig
