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Description
We employed typical acute pancreatitis models in vivo and in vitro and found that VB 12 activated CBS-SIRT1 axis to suppress oxidative stress and repair mitochondrial damage in the pathological process of acute pancreatitis
Studies have consistently reported that inhibiting NNMT activity or downregulating NNMT expression leads to significant increases in NAD + levels in mouse adipocytes (Neelakantan et al., 2018) and hepatocytes (Song et al., 2020)
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