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Next, invest in contact-site biology by creating tools and reference atlases that resolve microdomain redox at organelle interfaces (MAMs, peroxisomemitochondria, mitochondrialysosome) and plasma-membrane nanodomains, because that is where signalling turns into damage (Xia et al
379 The second possible mechanism is the autoantigen-like reaction triggered by the Atopobium antigen described in rheumatoid arthritis (RA)
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Roles of MiRNAs and SIRT1 in GC A 2021 study by Luo et al
