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in the form of iron sulfur clusters, iron enables electron transport to drive oxidative phosphorylation and energy production
However, considering that the hiPSC-derived neurons are cultured in the presence of a higher concentration of glutamine (2 mM) than that used during the incubation experiments (0.5 mM), it is plausible that increased glutamine metabolism in FTD3 neurons is a compensatory mechanism to fuel mitochondria and potentially accelerate ROS production
It is often activated by an infection or stress, but the antigen that triggers the disease is still unknown
