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It often involves autosomal dominant mutations in genes such as amyloid precursor protein (APP), presenilin 1 (PSEN1), and presenilin2 (PSEN2), which accelerate amyloid-beta accumulation and early pathological changes
Nigdy bezporednio na liofilizat, silny strumie moe zaburzy kompleks GHK-Cu
doi: 10.1016/j.jmwh.2010.06.018
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