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Description
In vitro experiments in NIH-3T3 cells revealed that the combination treatment downregulated -SMA expression and inhibited JAK2, STAT3, and STAT1 signaling pathways, reducing fibroblast activation and migration induced by TGF-1 and IL6, thereby suppressing fibrosis progression through the JAK2-STAT3/STAT1 pathway
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10.1067/mlc.2000.105976 207 MinJ
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