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N-terminal modification of TGF1 by SF
The oxidative stress-induced weakening of the fibrous cap covering the plaque, due to the degradation of extracellular matrix components by matrix metalloproteinases (MMPs), is a key factor in plaque instability [75]
(108) Chitosan-coated human serum albumin NPs can also interact with negatively charged cell membranes, subsequently crossing into hCMEC/D3 cells, accompanied by reversible and transient tight junction opening between the cells
showed that metastasis-associated fibroblasts (MAF) expressed high levels of CD38 and activated macrophages into a pro-tumoral M2-like macrophage [52]
