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In summary, we found that PRKN gene expression contributes to the network of available thiols in the cell, including by parkins participation in glutathione recycling, which involves a reversible, posttranslational modification at select cysteines

Numerous studies using models of cerebral ischemia in rodents indicate that Liraglutide reduces the volume of the infarct zone, has a neuroprotective and antioxidant effect, promotes angiogenesis and increases the expression of VEGF in the area of cerebral ischemia [76,77,78], improves metabolic and functional recovery after stroke [79], and reduces neurological deficits [80,81,82]

Barrett, J., Lack, L

K., Lee, V