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The role of OS in ALS pathology, whether as a primary cause of disease or a secondary consequence, is now well recognized and many preclinical and clinical studies have consistently demonstrated that the disease is characterized by high levels of OS markers able to induce cellular damage, impaired cell signalling, and finally cell death [25, 86]
They found that the incidence of such aberrations decreased by between 17.9% and 30% compared to age-matched controls
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The authors concluded that intravenous BPC157 was welltolerated, but stressed that larger, longerterm trials are needed
