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It can be seen that the regulation of metabolism on oxidative stress takes place a major part in the pathophysiological process of ARDS

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Progressive massive pulmonary fibrosis, diffuse interstitial fibrosis, and collagen accumulation involved in the development of pulmonary alveolar proteinosis following nano-indium-tin oxide nanoparticles (ITO) exposure in rats were shown (Liu et al., 2022)
