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In liver disease, SOD activity can initially rise as an adaptive response (e.g., pediatric cholestasis) but often declines with progression to HCC

6a, b), strongly suggesting that the reduction in PM distribution of -SNAP M105I protein observed in hyh brain samples is mainly, if not exclusively, due to intrinsic alterations in the lipid-binding capacity of the mutant protein

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Sakurai R, Suzuki H, Ogawa S, Takahashi M, Fujiwara Y