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Description
During the pathogenesis of OA, JUN directly binds to the promoter of NCOA4 and initializes its transcription, increasing ferritinophagy degradation, leading to ferroptosis and extracellular matrix degradation in chondrocytes
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Some effects may be sustained long-term, while others may require ongoing or periodic treatments to maintain optimal results
Cells 12 , 2115 (2023)
