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When AKI occurs, the renal tubules are damaged and the glomerular filtration rate drops sharply, which results in an increase in serum creatinine and blood urea nitrogen (17)
Other reports indicate that excessive levels of NO, as a result, for instance, from BPC 157 activity, can be responsible for damage of mitochondrial ironsulfur enzymes and hence for inhibition of mitochondrial respiration through inhibition of cytochrome oxidase [154,155]
