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Not only does this result in reduced availability of NO for vasodilation, but ROS and RNS can also induce the proliferation of smooth muscle cells and stimulate the expression of cell adhesion molecules, mostly ICAM-1 and VCAM-1, that control the inflammation on the vascular level by leukocyte migration and adhesion [52]
7b) indicating selective TBI responses when NF-B is either repressed or pre-activated in astrocytes
