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In selected contexts, modulate ACSL4/LPCAT3 (PUFA loading into membranes) and LOX activity to reduce peroxidation substrates and enzymatic initiation, while reinforcing parallel anti-ferroptotic circuitsFSP1CoQ at the plasma membrane and DHODHCoQH 2 in mitochondriato buffer lipid radicals without abolishing physiological signalling (Mao et al

[DOI] [PMC free article] [PubMed] [Google Scholar] 60.Schilling S., Goelz S., Linker R., Luehder F., Gold R

Like autoimmune diseases, these conditions are characterized by oxidative stress and inflammation and inappropriate release of histamine in many cases

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