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By day 85, however, many subjects had developed a preference for binding the charge pair mutations in the TL1A Fab, and some lost nearly all ability to bind to the non-charge pair mutation containing domain ( Figure 4C )
Guillot TS, Shepherd KR, Richardson JR, Wang MZ, Li Y, Emson PC, et al
A deficiency in SLC40A1 or overexpression of TFR causes iron overload, thereby triggering ferroptosis
(PubMed) Yang J, Wang T, Yang J, et al
