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AfkarianMZelnickLRHallYNHeagertyPJTuttleKWeissNSet al
ALOX5-mediated ferroptosis acts as a distinct cell death pathway upon oxidative stress in Huntingtons disease
The NO + serves as an effective oxidizing agent and is responsible for the S -nitrosylation of protein thiol groups [1]
Recent studies highlight the novel oxidative stress avoidance mechanism mediated by lipopolysaccharidebinding protein accumulation in lipid droplets, which serves as a hub connecting stressoxidative stressmetabolic disordersobesity [120]
