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Description
In vitro pull-down assay showed that GSTP-S184D displayed a stronger affinity to bind HMGB1 than GSTP (WT), but GSTP-S184A did not combine with HMGB1, indicating that Ser184 phosphorylation was necessary for the association between GSTP and HMGB1 (Figures 5H,I)
Early clinical results suggest promise for improved efficacy and reduced toxicity compared to conventional chemotherapy
Turn-off labeled aptasensors typically involve fluorophore-quencher pairs brought into proximity by target-induced conformational changes (Zhao et al., 2018)
12 (half mustard form) could be produced by non-catalytic process
