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Cardiovascular disease In cardiovascular disease, Myc and Myc-related factor X (Max) upregulate the Gln transporters SLC1A5 and SLC7A5 and mitochondrial malate in pulmonary hypertension, thereby promoting glutaminolysis-induced right ventricular hypertrophy

A block in MMUT activity leads to accumulation of methylmalonic acid and other propionyl-CoA derived catabolites, which in turn have secondary effects on associated pathways and play a major role in the pathogenesis of MMA (3)

Dal Lago A, De Martini D, Flore R, et al

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