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These results suggest that targeting FSP1 or inhibiting its phase separation initiates ferroptotic cell death is a potential anticancer paradigm
This results in increased AREs expression and proliferation of glutathione (GSH), heme oxygenase-1 (HO-1), superoxide dismutase (SOD), and NAD(P)H: As demonstrated in the relevant literature, quinone oxidoreductase 1 (NQO1) (Xia et al

All features suggested the promises of pFeSAN as artificial enzymes for biomedical applications

Anjum et al., 1992)
