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Pereiro, I
It has been reported that macrophage-derived NTN1 drive the development of fibrosis through a mechanism involving remodeled adrenergic nerves and their secretory product noradrenaline and 1 adrenoreceptors (Gao et al., 2021)
Furthermore, the reactivity of the disulfide bond can be modulated by steric hindrance
These studies imply that chemotherapy drugs induce lipid peroxidation within tumors
