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Finally, many exhibit anti-fibrotic and cardiomyocyte-protective actions: they interfere with fibrosis-related pathways (TGF-/Smad, etc.), inhibit cardiac fibroblast activation, and reduce pathological deposition of extracellular matrix in the heart (Xiao et al., 2018

A.BranumA.et al (2006)

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Huberman suggests that substances increasing angiogenesis could logically enhance the risk of cancer
