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doi: 10.1017/S0022029919000384

BPC-157 has been studied in tendon transection models with consistent findings: accelerated collagen synthesis, improved tensile strength at the repair site, and faster return of functional loading

The significant increase in fissures observed in the PCPA-induced insomnia model group suggests that sleep disturbances may lead to notable changes in brain tissue morphology (Figures 8A,B)

It is conceivable that BPC-157 may exhibit some engagement with the NO mechanism, possibly providing a safeguard for the endothelium and perhaps encouraging angiogenic behaviors by fostering the development of new circulatory routes