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doi: 10.1590/0004-282X20130209 192

Lentivectors have been developed from primate lentiviruses, such as the wild-type human immunodeficiency virus type 1 and non-primate lentiviruses, such as the equine infectious anemia virus, by progressively removing most of the viral genes from the vector genome to limit the risk of producing replication-competent viral particles (Cronin et al

Furthermore, exogenous IGF-I or IGF-II can restore differentiation in the presence of TGF-, indicating that IGF-II may be effective in treating sarcopenia in which TGF- plays a pathogenic role[9]

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