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To test whether the MTS pathway was supporting the TR/GR-null livers, we treated mice with single-dose challenges by inhibitors of cystine uptake (sulfasalazine, SSZ), possible cysteine uptake (2-aminobicyclo-(2,2,1)-heptane-2-carboxylic acid, BCH), conversion of methionine into cysteine by MTS (propargylglycine, PPGG, which inhibits trans -sulfuration and not the methionine cycle), or synthesis of GSH (BSO) 11

caldus MTH-04 We found a putative glutathione reductase encoding gene F0726_RS04210 in the genome of A

The fact that AtGrxS15 could not reduce oxidized roGFP2 with the help of GSH might point to an altered or blocked glutathione activator site

Particularly valuable during times of elevated oxidative stress