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T., Odden, M

doi:10.1016/0955-0674(91)90170-4

The results showed that mice from the maternal diabetic (STZ/P-VEH) group had significantly increased superoxide anion release (see Figure 5D) and 8-oxo-dG formation (see Figure 5E) compared to the control (CTL/P-VEH) group, and amygdala infusion of OXTR (STZ/P-OXTR) had no effect, while amygdala infusion of ER (STZ/P-ER) completely reversed the diabetes-mediated effect

The exact mechanism of eNOS modification is unknown but in vivo experiments have shown that eNOS activation in aortas and iNOS transgenetic expression in mouse heart both result in NO-induced protein S-glutathionylation