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That said, reviews are overwhelmingly positive
The progression from a completely normal artery to a clogged or atherosclerotic one follows a very clear path: an apoB containing particle gets past the endothelial layer into the subendothelial space, the particle and its cholesterol content is retained, immune cells arrive, an inflammatory response ensues fixing the apoB containing particles in place AND making more space for more of them
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Multiple caf au lait macules, however, can be associated with the autosomal dominant genodermatoses, neurofibromatosis type 1 and type 2 (NF1 and NF2, respectively)
