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Secondary Mechanisms: FAK-Paxillin and Growth Hormone Receptor Beyond the two primary pathways, published studies have identified additional mechanisms relevant to specific tissue types

An additional, 11 non-herniated human discs were used to examine causality: half of each specimen was cultured in a manner that allowed free unconstrained swelling (similar to a herniated disc in vivo), while the other half was cultured within a perspex ring that allowed constrained swelling

Researchers continue to study its effects on angiogenic signaling, fibroblast activity, extracellular matrix regulation, and cell migration to better understand the molecular mechanisms governing tissue maintenance and regeneration

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