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l-carnitine atherosclerosis intake and high trimethylamine N-oxide plasma levels correlate with low aortic lesions in ApoE−/− transgenic mice expressing CETP Revisiting the Role of Carnitine

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Abstract Dysregulation of lipid metabolic homeostasis is a central pathological feature of metabolic disorders, including obesity and non-alcoholic fatty liver disease

l-carnitine atherosclerosis intake and high trimethylamine N-oxide plasma levels correlate with low aortic lesions in ApoE/ transgenic mice expressing CETP Revisiting the Role of Carnitine

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l-carnitine atherosclerosis intake and high trimethylamine N-oxide plasma levels correlate with low aortic lesions in ApoE/ transgenic mice expressing CETP Revisiting the Role of Carnitine

If the differences between two consecutive measurements at the same position were greater than 10%, they were tested again after a 5 minute interval

l-carnitine atherosclerosis intake and high trimethylamine N-oxide plasma levels correlate with low aortic lesions in ApoE/ transgenic mice expressing CETP Revisiting the Role of Carnitine

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l-carnitine atherosclerosis intake and high trimethylamine N-oxide plasma levels correlate with low aortic lesions in ApoE/ transgenic mice expressing CETP Revisiting the Role of Carnitine

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