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The fact that wild-type ATXN3 binds to components of the transcriptional machinery in cells, as well as the transcriptional changes reported in animal models of SCA3 in the presymptomatic phase, favors the theory of a general transcriptional dysregulation as an initiating factor of SCA3 [117,118,119]

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That indicated poor red blood cell production, and those that were produced were malformed and had short life spans

[oai_citation:0PMC]( Key pointMost findings below come from in vitro or non-human in vivo experiments