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Natural compounds can modulate immune surveillance by enhancing NK cell activity, promoting dendritic cell maturation, and shifting macrophage polarization toward a tumorsuppressive M1 phenotype (cited [83, 84, 85, 146, 154])

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Though only a fraction of total CHAC1 is associated with the mitochondria in our cells, we show that mitochondrial CHAC1 supports the matrix cysteine pool and is sufficient to sustain Fe-S protein activity under cystine starvation