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Augmenter of liver regeneration protects the kidney from ischaemia-reperfusion injury in ferroptosis

Given this information, emerging therapies which target GSH homeostasis such as N-acetylcysteine (NAC), GSH esters, or activation of antioxidant response elements may provide new avenues to interrupt oxidant-driven inflammation in these diseases

Reduces Inflammatory Cytokines TB-500 decreases the production of pro-inflammatory mediators like IL-1, TNF-alpha, and other cytokines that drive excessive inflammation at injury sites

For example, increased levels of GSH and GSH related systems in cancer have been employed as a basis for anticancer prodrug activation, anticancer drug delivery and anticancer drug development