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Mechanisms of CVD and CKD in patients with diabetes Vascular damages of ASCVD in patients with diabetes are characterized by endothelial and vascular smooth muscle cell dysfunction [12]
Excess iron may promote the polarization of macrophages towards the M1 phenotype ( Figure 2 )
For example, the VPA-induced rat model is characterized by reduced oxytocin mRNA levels, fewer oxytocin-immunoreactive cell numbers in the paraventricular nucleus, and lower oxytocin concentration in the cerebrospinal fluid [31]
