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GCN2-dependent phosphorylation of eIF2 normally inhibits formation of this complex, but recent work shows that ATF4 target gene expression is also modulated by MR through a mechanism independent of both GCN2 and eIF2 27,28
Recurrent symptomatic VTE was similar between groups (1.1% vs
miRNAs sharing the same first 13 nt were collapsed into a single entry, and the full names of the collapsed species are provided in Supplementary Table 5
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