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[146] further reported that curcumin-derived CQDs inhibited amyloid-b fibril aggregation and the associated oxidative stress in AD models via interactions with b-sheet domains, preventing peptide self-assembly, and concurrent curcumin-mediated free-radical suppression [146]

A very high response efficiency may cause early and undesired drug activation, whereas a low response efficiency may induce no pharmacological actions

10.1038/nrendo.2010.123 Nat

First, the sulfonation reaction saturates and that allows more NAPQI to accumulate ([7, 24]) and second, the increased amount of NAPQI exhausts the liver stores of reduced glutathione (GSH)[6] as well as the livers capacity to synthesize new GSH