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For example, in A142 induced neurotoxicity, TQ protected cholinergic hiPSC neurons by restoring intracellular antioxidant levels and by inhibiting ROS generation, cell death, and apoptosis [51], and subsequently reduced quantal size of ready to release synaptic vesicle pools caused by A142 [51]
The first of these is statistical in nature, and is illustrated by considering the dipeptide Ala-Gly as a proposed target
They are quality-of-training improvements that compound over time
(1)-(5), are as specified in Table 3
