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A number of case series describing HSCT in the treatment of isolated CNS relapse have been published.[222,223] Although some reports have suggested a possible role for HSCT for patients with isolated CNS disease with very early relapse and T-cell disease, there is less evidence for the need for HSCT in early isolated CNS relapse in B-cell disease, and no evidence in late relapse

Pathologically activated astrocytes exhibit disrupted intracellular signaling or metabolic homeostasis, impairing EAAT2 transcription, translation, or trafficking

Monitoring Run baseline labs and follow-up blood work with attention to IGF-1 and metabolic markers relevant to growth-hormone-related research