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[DOI] [PMC free article] [PubMed] [Google Scholar] 130.Krishna D.N.G., Philip J
An example is treatment with the toxic bile acid lithocholic acid, which caused initial GCLC induction, followed later by suppression at the transcriptional level in hepatocytes [90]
Extracellular acidosis modulates drug block of Kv4.3 currents by flecainide and quinidine
The absence of suitable human models has also hindered mechanistic studies of -cell identity loss in T2D, a limitation now addressed by human embryonic stem cell (hESC)-derived islets (SC-islets), which are increasingly used to investigate human -cell development and islet pathology in diabetes
