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It was shown that NO produced by iNOS is able to inhibit the up-regulation of this pathway in rat lung macrophages activated with lipopolysaccharides (LPS) and interferon-gamma (IFN-), functioning as an immunosuppressor [43]

Front Oncol 9:873

In order to address the issue of therapeutic resistance driven by reprogramming of tumor glutamine metabolism, future studies must thoroughly examine the heterogeneity of glutamine dependence in different genetic backgrounds (e.g., KRAS-mutant vs