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Emphasizing the significance of an AD-specific neuroprotective peptide HN in mitigating AD-related neurotoxicity by activating the JAK2/STAT3 signaling axis, these researchers observed that the age- and disease-dependent decline in the JAK2/STAT3 axis plays a pivotal role in the pathogenesis of AD
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Finally, the authors observed that p-JNK and p-38 levels were reduced in 3-HAA-treated DC2.4 cells and bone marrow-derived DCs
