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The 47kDa Akt substrate associates with phosphodiesterase 3B and regulates its level in adipocytes
Therefore, there will not be any thermodynamic push to initiate the crystal growth [21, 35]
Overall, dysregulation of apoptosis in HT reflects both enhanced activation of extrinsic and intrinsic death pathways and suppression of survival signals, ultimately leading to irreversible thyroid tissue loss
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