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Conclusion The present study indicated that dietary L-carnitine supplementation could increase the concentrations of carnitine in the liver and muscle, and decrease the lipid content in both tissues, possibly through enhanced mitochondrial -oxidation and reduced adipogenesis

Physiological values of ROS are required to sustain the signaling pathways controlling crucial cellular processes, among which are inflammation, differentiation, proliferation, homeostasis, and apoptosis, whereas the generation of dysregulated ROS can amplify these same pathways, thus fostering disease phenotypes associated with ROS, as seen in type II diabetes, cancer, and atherosclerosis [19]

An acetyl-L-carnitine switch on mitochondrial dysfunction and rescue in the metabolomics study on aluminum oxide nanoparticles

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