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Collectively, l-Carnitine treatment reverses PFOS-mediated oxidative stress in cytosol and mitochondria through its antioxidative activity and subsequently protects RTCs from autophagy-associated apoptosis

After the metabolism measurement, the expression levels of LCFA transport proteins (FAT/CD36, CPTI) were analyzed in skeletal muscle

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Going forward, it will be of primary importance to better understand the in vivo metabolic profiles and dynamics in the kidneys and nephron segments of animals and humans, and the investigation of genetic and environmental factors that lead to the development of hypertension by influencing these metabolic processes may help identify any pro-hypertensive regulatory dysfunctions that result from such metabolic abnormalities