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Studies using in vitro and in vivo models of AD have shown that targeting oxidative stress has a positive effect on neural degeneration, inflammation, A1-42 aggregation, p-tau formation, GSH levels, iron overload, mitochondrial function, motor dysfunction and learning and memory (Fu et al., 2006

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doi: 10.1042/BCJ20160103

Crucial stages of ferroptosis include the accumulation of iron, lipid peroxidation, and GPX4 inactivation, all of which contribute to intestinal oxidative damage due to iron overload, leading to lipid peroxidation accumulation and potentially triggering the onset of IBD [63]