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After skin damage, the newly growing epidermis and skin cells at the wound edge express more metallothionein than does the surrounding normal skin

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HO-1 and HO-2 have roles in heme catabolism, but the role of HO-3 has not been well elucidated.15,16 HO may be inhibited by a number of metalloporphyrins, a phenomenon which has a therapeutic potential.17 In the fetus, microsomal HO activity in the liver may be as much as eight times higher than in the adult.18 This suggests that the increased catabolism of heme in neonates may be associated with an increased transcription of the inducible HO isoform, HO-1