α-Lipoic Acid and Cardiovascular Disease
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For a subset of genes, AR binding by low doses of doxorubicin increased at sites that harbored binding motifs for prostate-specific factors, such as FOXA1, supporting that AR dependence on cofactors at half sites may be the basis for differential modulation by doxorubicin [67]
These mechanisms include oxidative stress, impaired lipid metabolism, and persistent inflammation
Source : Tommi Raij, Kari Raij
